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Question

Ms. X. is showing increasing PVCs on the ECG. State the cause and describe the effect if these continue to increase in frequency.

On day 6 after admission Ms. X is preparing to go home with her son and they receive instructions on lifestyle modifications that are desirable if Ms. X is to avoid another MI. What measures should be included in such a discussion?

Discuss how stress could have played a factor in Ms. X’s MI and her future post discharge.

Ms. X’s condition becomes less stable and she remains in the hospital. On the seventh day following admission, she is found unconscious on the floor of her bathroom. Her pulse is weak and elevated, and her skin is moist with pallor evident. Her BP is 50 systolic. A diagnosis of cardiogenic shock is made, and resuscitation efforts are started.

Explain why Ms. X. has experienced cardiogenic shock at this time.

Describe the effects of cardiogenic shock on the organs of the body.

What problems will occur if decompensated shock occurs? How is compensation limited in this situation?

Ms. X dies shortly later. What is the cause of death in this case?
Step 1/3

There are many reasons why premature ventricular contractions might happen, including

Medication (antihypertensive, antidepressants) (antihypertensive, antidepressants)
excessive production of adrenaline
a rise in calcium levels, a fall in potassium, and a fall in magnesium
drinking and smoking
caffeine stress cardiovascular diseases
The patient's life may be in jeopardy if the PVC is continuous and frequent since it might cause arrhythmia and cardiomyopathy.
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The discussion or health education that follows must be conducted.
ANS.
Reduce stress
Eat less caffeine.
No drinking or smoking, if you do
medication Relaxation therapy (antihypertensive, beta blocker, anti arrhythmic drugs and calcium channel blocker)
lifestyle adjustment

Step 2/3

Cardiogenic shock is a physiological condition wherein systolic cardiac failure leads to insufficient tissue perfusion. Effects of Cardiogenic Shock on Organs of the Body Essay Paper

Ms. X's condition was unstable for six days. although it's unclear what caused it. Low cardiac output due to significant left ventricular (LV) infarction is typically the cause, which is then followed by a mechanical problem like a ventricular septal defect, papillary muscle rupture, or right ventricular (RV) infarction.
If it is caused by a cardiogenic factor, the pathophysiology would be brought on by low systolic pressure, which would result in less blood being evacuated from the ventricle. Therefore, as end-systolic volume rises, diastolic filling also rises, increasing LV end-diastolic pressure. However, this effort to boost cardiac output raises myocardial oxygen demand and may cause pulmonary oedema. The patient experiences increased left ventricular and right ventricular filling pressures as well as poor cardiac output as a result of impaired contractility. Because of the increased tissue oxygen extraction brought on by the decreased cardiac output and mixed venous oxygen saturation decline, there is a significant amount of arterial oxygen desaturation.

Explanation for step 2

The patient experiences increased left ventricular and right ventricular filling pressures as well as poor cardiac output as a result of impaired contractility.

Step 3/3

Acute systemic hypoperfusion causes tissue hypoxia and organ failure during shock. Reduced blood supply to end organs results in cellular hypoxia and damage to those tissues. The brain, heart, and kidney are the major organs that are impacted. Reduced cerebral perfusion brought on by a decreased cardiac output causes a variety of altered mental states, from agitation and bewilderment to flaccid coma. Hypoperfusion and increasing heart dysfunction are caused by low coronary perfusion. Oliguria and consequent renal failure are caused by impaired glomerular filtration as a result of the kidneys compensating for decreased perfusion.

Expert Answer

Question

Ms. X. is showing increasing PVCs on the ECG. State the cause and describe the effect if these continue to increase in frequency. On day 6 after admission Ms. X is preparing to go home with her son and they receive instructions on lifestyle modifications that are desirable if Ms. X is to avoid another MI. What measures should be included in such a discussion? Discuss how stress could have played a factor in Ms. X’s MI and her future post discharge. Ms. X’s condition becomes less stable and she remains in the hospital. On the seventh day following admission, she is found unconscious on the floor of her bathroom. Her pulse is weak and elevated, and her skin is moist with pallor evident. Her BP is 50 systolic. A diagnosis of cardiogenic shock is made, and resuscitation efforts are started. Explain why Ms. X. has experienced cardiogenic shock at this time. Describe the effects of cardiogenic shock on the organs of the body. What problems will occur if decompensated shock occurs? How is compensation limited in this situation? Ms. X dies shortly later. What is the cause of death in this case?

Step 1/3

There are many reasons why premature ventricular contractions might happen, including

Medication (antihypertensive, antidepressants) (antihypertensive, antidepressants)
excessive production of adrenaline
a rise in calcium levels, a fall in potassium, and a fall in magnesium
drinking and smoking
caffeine stress cardiovascular diseases
The patient's life may be in jeopardy if the PVC is continuous and frequent since it might cause arrhythmia and cardiomyopathy.
ORDER A PLAGIARISM-FREE PAPER HERE

The discussion or health education that follows must be conducted.

ANS.

Reduce stress

Eat less caffeine.

No drinking or smoking, if you do

medication Relaxation therapy (antihypertensive, beta blocker, anti arrhythmic drugs and calcium channel blocker)

lifestyle adjustment

Step 2/3

Cardiogenic shock is a physiological condition wherein systolic cardiac failure leads to insufficient tissue perfusion. Effects of Cardiogenic Shock on Organs of the Body Essay Paper

Ms. X's condition was unstable for six days. although it's unclear what caused it. Low cardiac output due to significant left ventricular (LV) infarction is typically the cause, which is then followed by a mechanical problem like a ventricular septal defect, papillary muscle rupture, or right ventricular (RV) infarction.

If it is caused by a cardiogenic factor, the pathophysiology would be brought on by low systolic pressure, which would result in less blood being evacuated from the ventricle. Therefore, as end-systolic volume rises, diastolic filling also rises, increasing LV end-diastolic pressure. However, this effort to boost cardiac output raises myocardial oxygen demand and may cause pulmonary oedema. The patient experiences increased left ventricular and right ventricular filling pressures as well as poor cardiac output as a result of impaired contractility. Because of the increased tissue oxygen extraction brought on by the decreased cardiac output and mixed venous oxygen saturation decline, there is a significant amount of arterial oxygen desaturation.

Explanation for step 2

The patient experiences increased left ventricular and right ventricular filling pressures as well as poor cardiac output as a result of impaired contractility.

Step 3/3

Acute systemic hypoperfusion causes tissue hypoxia and organ failure during shock. Reduced blood supply to end organs results in cellular hypoxia and damage to those tissues. The brain, heart, and kidney are the major organs that are impacted. Reduced cerebral perfusion brought on by a decreased cardiac output causes a variety of altered mental states, from agitation and bewilderment to flaccid coma. Hypoperfusion and increasing heart dysfunction are caused by low coronary perfusion. Oliguria and consequent renal failure are caused by impaired glomerular filtration as a result of the kidneys compensating for decreased perfusion.

"decompensated shock symptoms":

Systolic blood pressure is decreasing.
tachypnea and tachycardia
low urination
uneven or laboured breathing
body temperature being lower than normal, cyanosis, a thready or nonexistent peripheral pulse, and dilated pupils
decline in mental state

with decompensated shock (shock become worse), advanced life support is needed to rescue a patient. and priority should be given to airway and underlying cause of shock. If the condition remains untreated, shock becomes irreversible and leads to the death of the patient.

Final answer

A rapid response is a key to the successful treatment of shock, requires a speedy assessment and quick transport to emergency care. Effects of Cardiogenic Shock on Organs of the Body Essay Paper

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